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Regular aerobic exercise protects against impaired fasting plasma glucose-associated vascular endothelial dysfunction with aging

The proposed mechanism involves controlling glutamate release in the synaptic cleft, modulating NMDA receptor stimulation, affecting excitatory amino acid transporter (EAAT)-mediated reuptake, and maintaining the glutamate/glutamine cycle within astrocytes

By choosing the appropriate injection site and following established clinical guidelines, healthcare providers can reduce the risk of injury and ensure safe, effective medication administration

N-Methyl-D-Aspartic acid suppresses Akt activity through protein phosphatase in retinal ganglion cells