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We have presented the mechanisms of action and pathophysiology of AD review, which examines the advancements achieved in this field over the past ten years

Although it has been generally accepted that acquisition of a toxic property by mutant SOD1 forms rather than loss or gain of its superoxide scavenging activity is responsible for the manifestation of ALS phenotype, how exactly SOD1 mutations lead to the selective motor neuron death is not yet entirely clear

Some resemble those seen in LS, with focal and bilateral symmetrical lesions in the basal ganglia, thalamus, and brain stem, while others show brain atrophy or underdeveloped myelin

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