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The functioning and regeneration of some Prxs is an interesting example as it involves the formation of several cysteine oxidation forms that divert the catalytic role into H 2 O 2 signaling functions

We are currently investigating potential therapies in not only IPF, but also progressive pulmonary fibrosis (PPF), which is the clinical designation to describe patients who have ILD with a progressive fibrotic phenotype

Defects in formation of FANCD2 nuclear foci after cisplatin treatment can be used to indicate cisplatin sensitivity [17] and concomitant overexpression of PARP1 , FANCD2 , and TP53 predicted increasing resistance to platinum [18]

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