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This experiment clearly confirmed that recombinant PHF10-DPF binding to H3 is strongly agonized by H3K9AcK14Ac in contrast to the SMARCB1-WHD, as has been reported for the bromodomain of Swi2/Snf2 and the DPF domains of SWI/SNF accessory subunits DPF2 and DPF3 (Supplementary Fig

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Iron chelation therapy with DFO, DFP and DFX has been successful in reducing excessive iron accumulation, decreasing peroxidation of lipid and improving activity of mitochondrial cells (Grolez et al., 2015)

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