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Since linear tetrapyrroles, such as bilirubin, play a significant role as antioxidant and anti-inflammatory agents, the reduction of protoheme synthesis by B6 unavailabilty and its further degradation to bilirubin allows further vulnerability to inflammation and oxidative stress

Studies have indicated a significant decrease in steroid hormone synthesis in models with Beclin1 or Atg5 deletion, primarily attributed to a reduction in intracellular cholesterol supply [147, 148], suggesting that the autophagy-lysosome system may indirectly support steroidogenesis by preserving substrate availability [149]

The Journal of Biological Chemistry, 277, 2795327959