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doi: 10.1074/jbc.C100141200

These processes include the creation of ROS, disruption of mitochondrial function, and the activation of necrotic/apoptotic pathways, ultimately resulting in permanent excitotoxic damage to the neurons

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Preclinical evidence from the Sikiric lab and collaborators describes protective effects against ethanol-induced gastric lesions, NSAID-induced enteropathy, and ileoileal anastomotic disruption in rat models, with the latter studied under the Pliva clinical-development code PL-14736 (Tkalcevi et al., 2007