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The present findings are therefore consistent with prior evidence suggesting dysregulated amino acid metabolism in at least a subset of individuals with ASD, while also underscoring the metabolic diversity observed across the spectrum [16, 17, 30, 31]

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We show that pharmaceutical downregulation of CPT1 results in a slower disease progression when applied in the early phase of the disease, and that it has effects on several aspects of the disease phenotype, including motor and non-motor behavior