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Using intracranial injections of a lentiviral short hairpin RNA (shRNA) construct to lower Gsto2 selectively in iSPNs of Trkb mutant mice prevented the dopaminergic dysfunction, improved energy metabolism, and other molecular rescues

In fact, early treatment with riboflavin was reported to have reduced the severity of symptoms in an 8-year-old child with a homozygous nonsense mutation in FLAD1, that showed aberrant biochemical results in the newborn screening test for MADD [75]

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