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Subsequently, DNA sequence analysis identified GHS-R1a as a GPCR, and in situ hybridization revealed that it is expressed in the pituitary gland, as well as in the hypothalamus and hippocampus (Bennett et al., 1997

Following cellular uptake via endocytosis or phagocytosis, MNPs can disrupt membrane integrity and induce mitochondrial and lysosomal dysfunction, which activate inflammation-related signaling pathways, including NF-B and the NLRP3 inflammasome [31,32]

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"Effect of N -acetyl-cysteine in prevention of noise-induced hearing loss: a systematic review and meta-analysis of randomized controlled trials"