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doi: 10.1111/acel.2013.12.issue-5 125 WatadABragazziNLAdawiMAmitalHToubiEPoratB-Set al
In addition, A deposition and excessive tau phosphorylation in the brains of AD patients can further inhibit the expression of SLC7A11, exacerbating GSH depletion and GPX4 inactivation, and forming a vicious cycle of oxidative stress-ferroptosis-AD pathological damage
IGF1Ec expression in MG-63 human osteoblast-like osteosarcoma cells
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