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Description
In amyloid precursor protein plus presenilin-1 (APP/PS1) mice, Nrf2 overexpression caused by antioxidant therapy has been demonstrated to improve neuronal apoptosis, increase thioredoxin-1 (TRX-1), and prevent the formation of NLRP3 inflammatory bodies [109]
Basically, the translocation of MOTS-c from the mitochondria to the nucleus turns off the coenzyme that wants to store sugar creating a state of fat burning while also encouraging healthy mitochondria turnover
The association between autoimmune thyroiditis, autoimmune gastritis and type 1 diabetes
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