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Lastly, N-homocysteinylation of proteins is one process responsible for homocysteine toxicity, since it causes structural and functional loss
However, the role of specific metabolites in the pathogenesis of OSA still needs further clarification
Another explanation could be that these adducts may break into lactone and the substrates (for example, secondary amines) of former Micheal-type addition reaction, due to the reversibility of this type of reaction (Lawrence et al., 2001)
In a human neuroblastoma SH-SY5Y cell line overexpressing the A53T mutant -synuclein, Jiang et al
