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It shows its action by interacting with the autoinhibitory loop of c-MET as well as take advantage of small ATP binding site of c-MET to have greater specificity against the kinase molecule [113]

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It also improved the proliferative and secretory functions of fibroblasts and reversed morphological abnormalities of senescent cells (such as flattened cell shape, enlarged volume, and nuclear pyknosis)

Participants with hepatic impairment were recruited before those with normal hepatic function to help ensure similarity across groups in age and body weight