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Dimethyl fumarate improves cognitive deficits in chronic cerebral hypoperfusion rats by alleviating inflammation, oxidative stress, and ferroptosis via nrf2/are/nf-Kb signal pathway

Footnotes This article is part of a special issue entitled: Insulin Resistance, Diabetes and Metabolism published in Metabolism Open

Borg JJ

This ongoing cycle has two particularly powerful outcomes: generating methyl groups (especially for cellular repair such as DNA damage) and making glutathione (the bodys most powerful antioxidant)
