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doi: 10.1016/s0891-5849(97)00443-7
Petropoulos, S., Fernandes, ., Barros, L., & Ferreira, I
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Frataxin-knockout mouse myoblasts, heart, and skin fibroblasts all showed characteristics of ferroptosis, and Nrf2 was downregulated in Friedreichs Ataxia patients and frataxin-knockout mice, while Nrf2 activators EPI-743 or sulforaphane regulated redox imbalances and rescued ferroptosis in frataxin knockout cells
