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To view a copy of this license, visit About this article Cite this article Fronik, P., Gutmann, M., Vician, P
This makes GSH particularly important in the mitochondria in defending against both physiologically and pathologically generated oxidative stress [16,17]
This could be partly associated with the mitochondrial DNA (mtDNA) mutations in autistic children [16]
To determine whether p21mediated ROS induction was specific or also resulted from overexpression of other CDK inhibitors, we measured ROS levels in EJ cells, which express no wildtype endogenous p16, containing tetregulatable p16 (EJp16)
