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Description
Concurrently, stress-activated signaling nodes such as GATA2/NF-B/STAT3 and Yes-associated proteins (YAP)/TEA domain (TEAD) reinforce survival through cross-talk with epigenetic modifiers, stabilizing DTP phenotypes [39, 41, 42] (Table 3)
AndradeBBPavanKumar NMayer-BarberKDBarberDLSridharRRekhaVVet al
Beyond laboratory science, longitudinal epidemiological studies incorporating biomonitoring and mechanistic endpoints will be crucial to establish causal links between internal exposure and health outcomes
Ventzel L, Jensen AB, Jensen AR, Jensen TS, Finnerup NB
