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bark in MDA-MB231 breast cancer cells

However, in hypertensive patients, endothelial function is often compromised, leading to what is known as endothelial dysfunction. Typically, these cells modulate blood vessel dilation and contraction by releasing factors like NO, prostaglandins, and endothelin, thereby stabilizing blood pressure

GSH serves as a cofactor for GPX4, enabling it to reduce harmful lipid peroxides back to normal phospholipids and prevent ferroptosis [31]

Sirtuin regulates cigarette smoke-induced proinflammatory mediator release via RelA/p65 NF-kappaB in macrophages in vitro and in rat lungs in vivo: Implications for chronic inflammation and aging