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The mechanism involves the glycine receptor (GlyR), a ligand-gated chloride ion channel expressed on immune cells

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Potential mechanisms include: Alterations in methylation pathways Changes in NAD+ availability Mild dehydration secondary to metabolic shifts Headaches are generally mild but may become persistent in sensitive individuals

The Mycobacterium tuberculosis PhoPR virulence system regulates expression of the universal second messenger c-di-AMP and impacts vaccine safety and efficacy