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Similarly, in gastric ulcers, ALDH2 activation decreases inflammation and oxidative stress while suppressing ferroptosis-associated damage via inhibition of the nucleotide-binding domain, leucine-rich repeat and pyrin domain-containing protein 3 inflammasome (127)

& Garcia-Verdugo, J

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It is hypothesized that high glucose concentrations in red blood cell (RBC) preservatives lead to increased methylglyoxal (MG) metabolism, causing glycation-driven damage to transfused RBCs