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Neuroprotective mechanism of hypoxic post-conditioning involves HIF1-associated regulation of the pentose phosphate pathway in rat brain

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To this end, buthionine sulphoximine (BSO), which inhibits -glutamylcysteine synthetase 36 , was used to treat T315I-Bcr-Abl-32D cells

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Without controlled human data, it is impossible to separate real effects from placebo

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Accepted January 2013

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