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To elucidate the relationship between iron homeostasis and metabolic features in clinical cases of MASLD, we conducted a comprehensive analysis of serum ferritin, a marker of iron stores, and its associations with diverse metabolic parameters in a well-characterized MASLD cohort (Figure 1A)

Animal experiments further revealed that cerebral hypoperfusion triggers a series of molecular and cellular events: insufficient brain perfusion first leads to dysfunctional mitochondrial energy metabolism, subsequently inducing excitotoxicity (primarily mediated by the overactivation of NMDA/AMPA receptors), and ultimately resulting in neuronal dysfunction and death [427]

The GATM and GAMT genes are expressed in neural tissues

In addition, ischemia-reperfusion has emerged as a potential challenge in patients undergoing ovarian transplantation