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To counteract this imbalance, cancer cells adapt by upregulating GSH biosynthesis, maintaining a redox equilibrium that supports survival and proliferation (Traverso et al., 2013)

2f is rate-limiting and no enzymesubstrate complexes accumulate in contrast to enzymes with typical MichaelisMenten kinetics 2,39,52

This ROS-associated mitochondrial damage was consistently observed in our study

Previously reported RNA sequencing (RNA-seq) data 25 revealed a significant upregulation of astrogliosis markers, including glial fibrillary acidic protein (GFAP), monoamine oxidase B (MAOB), 18 aldehyde Dehydrogenase 1 Family Member L1 (ALDH1L1), acetaldehyde dehydrogenase 2 (ALDH2), and aquaporin 4 (AQP4) 19,26 (Supplementary Fig