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Description
These findings suggest that NADPH not only is central to the cellular redox balance but also plays a critical role in regulating ferroptosis, potentially by influencing iron metabolism and lipid peroxidation (101)

In addition, by activating the PI3K/Akt pathway and speeding up the translocation of NFE2-Nrf2 to the nucleus via KEAP1 degradation, hASC-EV treatment significantly decreased oxidative damage

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8a vs c)
