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Such transition was shown to be controlled by the Nuclear Receptor Subfamily 4 Group A Member 1 (Nr4a1), which crucially modulates both the early Ly-6C high monocyte inflammatory and the later Ly-6C low macrophage reparative phases in the infarcted myocardium (172)

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In addition, they have anti-inflammatory effects and can be used as acne-mitigating candidates for topical application (Park et al., 2004)