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CONCLUSION This review aimed to elucidate the principal mechanisms involved in the neuropsychiatric manifestations of COVID-19, which involve glutamate dysregulation, Xct, Nf-B, mGluR2, and mGluR3, astrocyte and glial damage and neurotoxicity highlighted as central factors [15], exploring the potential of NAC and ALC as promising therapeutic agents in neuropsychiatric conditions COVID-19-related, including those associated with PCS
These functions are primarily mediated through the melanocortin receptors (MCRs) in various tissues [2]

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