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Human and mouse single-nucleus transcriptomics reveal TREM2-dependent and TREM2-independent cellular responses in Alzheimers disease

characterizing critical developmental windows via longitudinal cohorts

Gram-negative and gram-positive bacteria, viral, fungal, and parasite agents also account for it

NLRP3 deficiency alleviates severe acute pancreatitis and pancreatitis-associated lung injury in a mouse model
