JCI
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Curcumin-induced cell death depends on the level of autophagic flux in A172 and U87MG human gliabiastoma cells
It has been proposed that the impaired clearance and increased release of glutamate by activated glial cells upregulates glutamate levels and disturbs signaling through ionotropic and metabotropic glutamate receptors, thus contributing to neuronal dysfunction and, ultimately, behavioral changes [129]
LeeGRJangSHKimCJKimARYoonDJParkNHet al
Click on the link given below: Primary Molecular Mechanisms BPC-157 does not work through a single cellular path
