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As potential therapeutics for MM, sdAbs such as WF211, MU1067, JK36, genetically fused to the hinge and Fc domains of human IgG1, have demonstrated the ability to elicit antibody-dependent cytotoxicity against CD38 + cancer cells [89]
Methods: In the in vivo experiment, C57BL/6J mice were exposed to CS for 12 weeks to induce pulmonary emphysema
Consumer social and psychological factors influencing the use of genetically modified foodsa review
The neurofibromatosis type 1 gene product neurofibromin enhances cell motility by regulating actin filament dynamics via the Rho-ROCK-LIMK2-cofilin pathway
