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Additionally, we explore the randomized controlled trials, guiding future preventive healthcare strategies and clinical practices
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Additionally, the accumulation of senescent glial and neuronal cells has been shown to drive the senescence-associated secretory phenotype (SASP), further amplifying inflammation and oxidative damage
Therefore, we employed in silico, in vitro and in vivo approaches to investigate whether the absence of a functional GSTM1 enzyme in a null variant can be compensated for by other family members
