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3a), among which, p53-pS15 was upregulated in keloid fibroblasts (FC = 1.3)

LH/FSH dysregulation an elevated LH-to-FSH ratio produced by abnormally rapid GnRH pulse frequency in the hypothalamus, creating a hormonal environment that stimulates androgen production while suppressing follicle maturation.[3] Chronic low-grade inflammation elevated CRP, IL-6, and TNF-, even in lean women with PCOS and in the absence of obesity, suggesting that inflammation is a core feature, not a secondary consequence of excess weight

Digital images were captured using an integrated imaging system to evaluate callose deposition levels

By attaching itself to its receptors, this hormone works in tandem with insulin to control blood sugar levels [14]