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doi: 10.1074/jbc.271.10.5414
These results highlight hepsin's crucial role in controlling APAP-induced liver toxicity by regulating the cellular abundance of GJ proteins, which can affect the transmission of toxic metabolites or cell death signals and potentially increase oxidative stress
gondii infection (RH) group ( n = 10 mice/group)
And with growing data backing its supportive role in multiple physiological systems, it continues to gain popularity among proactive, self-aware health consumers
