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Ni H, Zhao W, Kong X, Li H, Ouyang J

114 In the context of ALS, the interaction of CCS with mutant SOD1 is faulty, resulting in decreased delivery of Cu to the mitochondria and the accumulation of the less-stabilized SOD1, which is prone to acquire pro-oxidants, consequently causing a toxic gain-of-function effect in motor neurons

From a pharmacokinetic perspective, the ethyl ester modification in N-Acetyl-L-cysteine Ethyl Ester significantly improves its ability to cross membranes, including the blood-brain barrier, resulting in potentially higher bioavailability than standard NAC

Two hundred proof ethanol (catalog #A405P4) was purchased from Fisher Scientific (Hampton, NH)