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Expression of iron homeostasis proteins in the spinal cord in experimental autoimmune encephalomyelitis and their implications for iron accumulation

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This dynamic interaction between inflammation, oxidative stress, and BBB breakdown contributes significantly to the progression of neurodegenerative diseases such as AD and PD and underscores the importance of targeting both oxidative stress and inflammation in therapeutic strategies [56,144,196]

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Instead AP-1 appears to be essential for TBH-mediated induction of the rat GCLC gene (Yang et al., 2002)

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