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Generation of 2A -adrenergic receptor knockout cell line We employed a CRISPR-Cas9-mediated approach to delete endogenous 2A -adrenergic receptor expression in INS-1E cells
These latter processes contribute to the neuronal inflammation response a key factor in neurodegenerative diseases including AD [34-36]
On Day 0, each rabbit underwent a surgical procedure to create two full-thickness open wounds
Meaningful disc recovery requires ongoing assessment and willingness to adjust the approach based on response
