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Hellwege, J

Mechanistically, SLC25A39 loss impaired mitochondrial oxidative phosphorylation, increased reactive oxygen species (ROS), and triggered apoptosis

One study has reported that resveratrol significantly reduced APAP-induced JNK activation and mitochondrial oxidative damage

The therapy counteracted both early and delayed neural hippocampal damage, achieving full functional recovery (Morris water maze test, inclined beam-walking test, lateral push test) [38], as assessed at 24 h and 72 h of the reperfusion
