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Knockdown of SLC1A5_var resulted in reduced GSH and increased ROS levels, while overexpression of SLC1A5_var promoted PC cell and tumor growth [36]

The clinical manifestations are diverse and can significantly reduce the quality of life for affected individuals [1, 2]

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Chronic heavy drinking induces the microsomal ethanol-oxidizing system (MEOS) and CYP2E1 enzyme upregulation, increasing ethanol clearance speed while simultaneously generating reactive oxygen species that accelerate hepatocellular damage