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Up-regulation of GPX4 was proven to depress ferroptosis in IS or HS, while excessive and lethal accumulation of lipid ROS in biofilms led to the depletion of GSH and inactivation of GPX4 (Jin et al

A better understanding of the etiology of this diagnosis will help identify those men likely to benefit from antioxidant therapy while minimizing the harmful effects of antioxidant overdosing

To determine CI activity (NADH-dependent OCR), MAS plus alamethicin (2.5 g ml 1 ) and cytochrome c (10 g ml 1 ) was added onto the cells followed by 5 mM pyruvate + 5 mM malate + 1 mM NADH + 10 nM recombinant perfringolysin O at port A, 2 M rotenone + 4 M antimycin A at port B, 0.5 mM TMPD + 1 mM ascorbic acid at port C and 50 mM azide at port D

Stabilit post-ricostituzione: Conservare a 2-8C al riparo dalla luce