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The upregulation of isocitrate dehydrogenase (IDH) [77], aconitase [78], and -ketoglutarate dehydrogenase (OGDH) [79] modulates ROS levels and cisplatin sensitivity, whereas succinate dehydrogenase (SDH) deficiency stabilizes HIF-1 activity and drives glycolysis-dependent resistance [80, 81]

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However, the heyday of thymalin was short-lived