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In turn, the impairment in -oxidation can shift the metabolism of VPA toward predominantly peroxisomal -oxidation, resulting in excessive production and accumulation of -oxidation products, including 4-en-VPA a metabolite that is incriminated in VPA-induced hepatotoxicity

2005 Jan;28(1):89-94

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contributed to the sections of the article describing the properties and molecular mechanisms of the antioxidant substances used in the treatment of the disease