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In addition, nNOS could indirectly enhance the activity of eNOS by increasing penile blood flow and shear stress, whose molecular mechanism is to activate the phosphatidylinositol 3-kinase (PI3K)/Akt pathway and consequently phosphorylate Ser 1177 of eNOS (Moreland, 1998

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Therefore, stabilization of mitochondria following antioxidant treatment could attenuate inflammatory processes and decrease the reaction of microglia and macrophages in the injured spinal cord