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Subsequent sections examine the core mechanismsanti-apoptotic, angiogenic, antioxidative, immunoregulatory and epigeneticthrough which MSCs reprogramme the damaged ovary, and we evaluate complementary strategies such as cell-free exosomes, small-molecule pre-conditioning and bio-engineered artificial ovaries
The remainder of the iron leaves the cell via membrane iron transport protein (FPN)
Channel 4s findings are the latest in a growing body of evidence pointing to major regulatory gaps that urgently need closing
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