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Description

A commonly agreed upon the fact in the pathogenesis of SLE is the dysfunction of T cells, B cells, dendritic cells, macrophages, and neutrophils, likely triggered by genes and/or the environment [4]

B.ShortS.MelcherA

Liu et al., 1996

However, l-serine supplementation does not reduce hyperglycemia in BKS- db/db models despite the improvement in neuropathy phenotypes, suggesting that some forms of diabetic peripheral neuropathy are more directly driven by l-serine deficiency as opposed to hyperglycemia