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The maximum of SI was between 3 h (CAR) and 6 h (CON), whereby CAR-supplementation had no significant effect

Due to TMAO excretion in urine [56, 57], in chronic renal disease patients, TMAO elimination from the body fails, causing elevation of its plasma concentration [78]

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As a result, immune cells such as TAMs, tumor-associated neutrophils (TANs), tumor-infiltrating T lymphocytes (TILs) and MDSCs can modulate cancer cachexia via the following mechanisms: (1) they impact key target organs such as skeletal muscle, adipose tissue and even the nervous system, inducing a cascade of pathological effects within these tissues through signal transduction mechanisms