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Treatment with 2012 did not produce a dose-dependent reduction in the viability of normal human epidermal melanocyte (NHEM) cells, normal human hepatic LX-2 cells, or human hepatoma Hep-G2 cells (Fig
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On this basis, we posit that the therapeutic action of Erastin in EMS is critically mediated by these ROS-driven programs, and that pharmacologic suppression of mitophagy may further sensitize lesions to Erastin
