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Pharmacological inhibition of mitochondrial -oxidation (e.g., with 4-bromocrotonic acid) or peroxisomal -oxidation (e.g., with thioridazine) under glucose-deprived conditions markedly compromises oligodendrocytes survival and optic nerve conduction, underscoring the central role of this catabolic pathway in white matter energy homeostasis
[DOI] [PubMed] [Google Scholar] 83.Board P.G., Menon D
Recently, significant advancements have been made in understanding vitiligo pathophysiology, with extensive characterization of the immunological bases (Seneschal et al., 2021
Increase your intake of vitamins C and E
