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doi: 10.3109/03009740903362511
It works by protecting cells from oxidative damage and inflammation, both of which are major contributors to the development of these conditions
Elevated lipid peroxidation markers (e.g., 4-HNE), protein carbonyls, and OGG1 levels in the SN of PD patients correlate positively with disease progression [226]
Recent evidence suggested that iron accumulated LPO triggered ferroptosis as a evolutionally conserved, regulated cell death pathway and mediated chondrocyte loss in OA
