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Heat shock response A rapid cellular response to stress that increases, usually via transcriptional activation, the availability of molecular chaperones to enable cytoprotection

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It is not unlikely that hyperglycemia-stimulated mitochondrial dysfunction and apoptosis account for cardiac metabolic memory [200]

Indeed, early studies have found that hydrochlorothiazide has the potential to influence various facets of glucose homeostasis, spanning from insulin secretion in the islets to the production of glucose in the liver and kidneys under diverse physiopathological conditions in humans and animal models