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Description
DASH collaborative research group
By selectively disrupting key antioxidant systemssuch as inhibiting glutathione synthesis with buthionine sulfoximine (BSO), blocking thioredoxin reductase with auranofin, or directly targeting NRF2therapies can push cancer cells beyond their limited oxidative buffering capacity
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Methylcobalamin is another non-prescription form popular in wellness settings, with more limited clinical evidence than hydroxocobalamin for treating true deficiency
