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In parallel, altered mechanotransduction and inflammatory signaling may activate NADPH oxidase isoforms, providing additional non-mitochondrial ROS sources

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This gain-of-function mutation drives the neoplastic transformation of neuroendocrine tissues and is transmitted through autosomal dominant inheritance

Despite recent advancements in evidence-based interventions for PTSD, many patients continue to suffer substantial residual symptoms post-treatment [13, 14, 38]
